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Figure 5. INT2–31 disrupts FAK-IGF-1R-dependent signaling and abrogates IGF-dependent AKT activation. Effect of increasing doses of INT2–31 in the presence and absence of IGF-1 (10 ng/dl) stimulation on signaling in (A) C8161 and (B) A375 cells after 24 h. All shown figures are representative of experiments performed in triplicate.
