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. 2012 Aug 3;303(7):H853–H862. doi: 10.1152/ajpheart.00154.2012

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Fig. 4. — Ca²⁺ transient characteristics are unaltered in UCD-T2DM rats at start of APAU treatment. A: Ca²⁺ transient amplitude in cardiac myocytes from prediabetic UCD-T2DM rats and control, nondiabetic rats. F/F₀, measured fluorescence signal (F)/fluorescence signal in resting myocytes (F₀). B: Ca²⁺ transient decay time (τ) in myocytes from prediabetic (pre-DM) UCD-T2DM rats and control, nondiabetic rats. C: diastolic intracellular Ca²⁺ concentration ([Ca²⁺]_i) in myocytes from prediabetic UCD-T2DM rats and control, nondiabetic rats. n > 20 myocytes from 4 different animals/group. The rats used had an average age of 15 ± 2 wk.