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Theoretical model of interactions between FGFR2, FGF, heparin, and vector in the absence of soluble heparin. (A) SASTG inefficiently binds to the undimerized form of FGFR2 and fewer viruses enter the cell. (B) With the addition of soluble heparin, SASTG is able to interact with the dimerized FGFR2 and enter the cell. Color images available online at www.liebertpub.com/hum
