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. 2012 Oct 23;79(17):1767–1773. doi: 10.1212/WNL.0b013e3182703f76

Figure 1. Tumor necrosis factor death receptor I (TNFRI) signaling is increased in Parkinson disease (PD) brains.

Figure 1

(A) ELISA showed that TNFRI levels are elevated in the temporal cortex of PD brains compared to the control brains. (B) Representative immunoprecipitation of tumor necrosis factor death receptor I–associated death domain protein (TRADD) was presented in the temporal cortex from parkinsonian brains and controls. (C) Spot density analysis of TRADD was normalized to corresponding β-actin level (ap < 0.05; bp < 0.01) compared to nonpathologic control (NPC).