Table 1. Genotype information and quality control statistics for the 9 obesity-susceptibility variants included in the study.

SNP	Chrom.	Position	Nearest gene	Effect allele	Effect allele	Other allele	References	Call rate	Genotype freq			HWE
					frequency				Risk allele homozygous	Heterozygous	Other allele homo	p-value
					%				%	%	%
rs2815752	1	72585028	NEGR1	A	59.2	G	1,6	99.7	34.5	49.3	16.2	0.525
rs6548238	2	624905	TMEM18	C	83.7	T	1,6	99.4	69.9	27.5	2.6	0.669
rs10938397	4	44877284	GNPDA2	G	38.8	A	1,6	99.4	15.2	47.3	37.6	0.765
rs987237	6	50911009	TFAP2B	G	17.7	A	9	98.4	3.3	28.8	67.9	0.745
rs10838738	11	47619625	MTCH2	G	36.3	A	1,6	99.6	13.9	44.7	41.4	0.221
rs4074134	11	27603861	BDNF	G	81.5	A	7,8	99.7	66.4	30.2	3.4	0.752
rs1121980	16	52366748	In FTO	A	44.4	G	1,3	99.7	18.9	51.0	30.1	0.139
rs17782313	18	56002077	MC4R	C	26.9	T	4	98.4	6.7	40.4	52.9	0.203
rs11084753	19	39013977	KCTD15	G	69.5	A	1,6	99.2	47.6	43.7	8.7	0.181

Article reference: 1) Loos et al., 2009; 2) Hinney et al., 2007; 3) Loos et al., 2008; 4) Willer et al., 2009; 5) Zhao et al., 2009; 6) Thorleifson et al., 2009;

HWE: Hardy-Weinberg equilibrium; Call-rate: rate of successful genotyping. All variants passed initial quality-control criteria with a call-rate ≥95% and genotype distribution were in Hardy-Weinberg equilibrium (P>0.05). The genotype distribution and effect allele frequencies varied from 17.7% for rs987237 to 83.7% for rs654238), which were in consistency with previous reports.