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. 2012 Oct 15;2012:438460. doi: 10.1155/2012/438460

Figure 5.

Figure 5

μ-opioid receptor agonist DAMGO abolishes the increase in NMDAR-mediated EPSC induced by low frequency stimulation at ST36. (a) Representative traces of NMDAR-mediated EPSC. (b) The increase of NMDAR-mediated EPSC in labeled DMV neurons induced by ST36 stimulation is reduced by bath perfusion of μ-opioid receptor agonist DAMGO and reversed by a specific μ-opioid receptor antagonist naloxonazine. *P < 0.05, compared to control; # P < 0.05, compared to the stimulated rats, n = 12–14, one-way ANOVA followed by q test. (c) Representative traces of NMDAR-mediated EPSC induced by paired-pulse. (d) The PPR decreased by ST36 stimulation is increased by DAMGO. *P < 0.05, n = 7-8, compared to ST36 stimulation and ST36 stimulation with naloxonazine, one-way ANOVA followed by q test.