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. Author manuscript; available in PMC: 2013 Sep 14.

Published in final edited form as: Circ Res. 2012 Jul 31;111(7):894–906. doi: 10.1161/CIRCRESAHA.112.273649

A, Human myocytes and vessels show human DNA sequences (Alu probe, white dots in nuclei) and human X-chromosome (X-Chr, single magenta dots in nuclei). B, hCSCs with old DNA had a more positive effect on left ventricular (LV) end-diastolic pressure (LVEDP), LV systolic pressure (LVSP), LV developed pressure (LVDP), positive and negative dP/dt, and calculated diastolic wall stress than hCSCs carrying the new DNA. C, Number of arrhythmic events in SO, and untreated and treated infarcts. For abbreviation and statistics see Figure 6B.

A, Human myocytes and vessels show human DNA sequences (Alu probe, white dots in nuclei) and human X-chromosome (X-Chr, single magenta dots in nuclei). B, hCSCs with old DNA had a more positive effect on left ventricular (LV) end-diastolic pressure (LVEDP), LV systolic pressure (LVSP), LV developed pressure (LVDP), positive and negative dP/dt, and calculated diastolic wall stress than hCSCs carrying the new DNA. C, Number of arrhythmic events in SO, and untreated and treated infarcts. For abbreviation and statistics see Figure 6B.