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. Author manuscript; available in PMC: 2013 Dec 1.
Published in final edited form as: Neuropharmacology. 2012 Sep 1;63(8):1327–1334. doi: 10.1016/j.neuropharm.2012.08.020

Figure 2.

Figure 2

Decreased KCl-evoked DA release in the SHR model of ADHD-C. A) The model of ADHD-C demonstrated significantly decreased KCl-evoked DA release versus the model of ADHD-PI in the dorsal Str (**p<0.01). The control SD also displayed decreased KCl-evoked DA versus the model of ADHD-PI in the dorsal Str (‡‡p<0.01). In the intermediate Str, it was observed that the control WKY displayed significantly increased KCl-evoked DA release versus the control SD strain (§p<0.05). Mean ± SEM. B) Representative tracing of the difference in KCl-evoked DA release in the dorsal Str between the model of ADHD-C, the model of ADHD-PI, and the control SD. C) Representative tracing of the difference in KCl-evoked DA release in the intermediate Str between the control SD and control WKY strains. Arrows indicate local application of KCl.