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. 2011 Apr 6;31(14):5335–5347. doi: 10.1523/JNEUROSCI.4811-10.2011

Table 2.

frac and Mmp2 genetically interact

Genotype Total defasciculated ISNba (n) Misprojection Hypofasciculation Hyperfasciculation Total defasciculated SNaa (n) Misprojection Hypofasciculation Hyperfasciculation
Mi{ET1}CG7526MB05690 13% (160) 3% 11% 0% 11% (152) 1% 11% 0%
Mmp2W307* 81% (133) 23% 79% 0% 42% (127) 5% 37% 0%
fracΔ1 zygotic 64% (119) 29% 51% 0% 49% (119) 9% 46% 0%
Mmp2W307*/+ 15% (140) 2% 13% 0% 5% (126) 0% 5% 0%
fracΔ1/+ 21% (160) 1% 19% 0% 6% (136) 0% 6% 0%
Mmp2W307*/+; fracΔ1/+ 46% (128) 18% 38% 0% 16% (122) 1% 16% 0%
repo>Mmp2b 3% (120) 0% 3% 63% 9% (109) 0% 9% 61%
fracΔ1, repo>Mmp2 47% (234) 3% 45% 3% 21% (215) 1% 20% 1%
24B>frac #3 63% (120) 16% 53% 0% 39% (115) 6% 33% 0%
24B>frac #7 57% (221) 17% 48% 0% 30% (214) 3% 28% 0%

n, The number of hemisegments scored for each genotype.

aHemisegments may have defects in both misprojection and hypofasciculation, yielding a total defasciculation that is not the sum of the two separate categories.

bData reported by Miller et al. (2008).