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. Author manuscript; available in PMC: 2013 Oct 1.
Published in final edited form as: Neurobiol Learn Mem. 2012 Sep 26;98(3):303–310. doi: 10.1016/j.nlm.2012.09.003

Figure 4.

Figure 4

The effect of DU-14 and vehicle treatment on retention crossover latency. Kruskal-Wallis analysis (p=0.05; h=7.92) shows that crossover latency was significantly longer in the lesioned DU-14 treatment group (n=6) compared to the lesioned vehicle group (n=8; * = p< 0.05 Dunn’s post-hoc test). There was a non-significant trend for an increase in retention crossover latency in the intact DU-14 treatment group (n=11) compared to intact vehicle (n=9).