Table 1.
Involvement of miR-133b in different physiological situations.
Situation | Reference |
---|---|
Translational regulation of utrophin: miR-133b, related to Duchenne muscular dystrophy, mediates the repression, and confirms repression of miR-206 | Basu et al. (2011) |
Formation of homologue clusters with miR-206: dysregulation role | Nohata et al. (2012) |
Upregulation during late stages of human, fetal muscle development | Koutsoulidou et al. (2011) |
When downregulated, miR-133b may have important implications in pathogenesis of essential hypertension | Yu et al. (2011a) |
Co-regulation of miR-133b with miR-206, novel biomarkers of Th 17-type immune reactions | Haas et al. (2011) |
Desregulation of miR-133b is associated with overall survival and metastasis in colorectal cancer | Akcakaya et al. (2011) |
Increase of miR-133b in mouse pectoralis muscle: regulation by myostatin | Rachagani et al. (2010) |
Upregulated miR-133b in mouse liver by tyrosine hormone | Dong et al. (2010) |
MiR-133b is upregulated on head and neck cancer | Liu et al. (2009) |
Mir-133b is regulated by endurance exercise in human skeletal muscle | Nielsen et al. (2010) |
Mir-133b is a biomarker of myocardial infection | D’Alessandra et al. (2010) |
MiR-133b targets prosurvival molecules MCL-1 and BCL262 in lung cancer | Crawford et al. (2009) |