Table 8. Studies of pesticide exposure and peripheral nerve function.

Study	Exposure state	Exposed population	Substance	Number	Comparison group	Findings
Boildin et al (1979) [6]	Acute	Adult cats	DFP (OP)	5	none	Focal distal non terminal axonal degeneration
Jayawardana et al (2008) [20]	Acute	Patients	OP	60	none	Normal sensory and motor nerve conduction
Steenland et al (1994) [21]	Acute on chronic	Farm workers	OP	83	Friends	Normal nerve conduction studies (Significant deficit was observed in nerve conduction velocity and/or amplitude among men poisoned by chlorpyrifos or phosalone)
Senanayake et al (1987) [22]	Acute	Patients with intermediate syndrome	OP	6	none	Normal nerve conduction
Senanayake et al (1982) [23]	Acute	Patients	Methamidophos	10	none	EMG denervation, normal nerve conduction
Kamel et al (2004) [8]	Review article	-	Pesticide	-	-	High-level exposure to OP can cause sensory abnormalities and motor dysfunction. But there was less evidence that low to moderate level exposure to OP was related to deficits in sensory or motor function or peripheral nerve conduction.
Steenland et al (2000) [9]	Chronic	Termiticide applicators	Chlorpyrifos	191	friends	Sensory neuropathy
Engle et al (1998) [10]	Chronic	Farm workers	OP	67	Matched reference subjects	Normal nerve conduction studies
Misra et al (1998) [12]	Chronic	Farm workers	Fenthion	24	Same subjects three weeks withdrawal from work	Reduction of peroneal MNCV, distal motor latency of median and peroneal
Ruijten et al (1994) [11]	Chronic	Farm workers	Mixed pesticide	131	Volenteers from the general population	Decreased motor nerve conduction velocity was found in median and peroneal nerves. Sensory nerve conduction velocity was found in median and sural nerves.