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. 2012 Dec 3;2:916. doi: 10.1038/srep00916

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Copyright © 2012, Macmillan Publishers Limited. All rights reserved

This work is licensed under a Creative Commons Attribution-NonCommercial-ShareAlike 3.0 Unported License. To view a copy of this license, visit http://creativecommons.org/licenses/by-nc-sa/3.0/

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HeLa cells were serum-starved for 24 h and then treated with or without 0.05–10 μM of U0126, a MEK inhibitor (A–C), or 20–150 μM of DPQ, a PARP-1 inhibitor (D–F), 1 h before stimulation with 100 nM of PMA for 10 h. A and B1 represent region A and B1 of the H1R promoter, respectively. , WT promoter; , MT1 promoter. Data are presented as mean ± S.E.M. [n = 4 (A, B, and D), 7 (C and F), or 3 (E)]. **, p < 0.05 vs. control.

Inline graphic — HeLa cells were serum-starved for 24 h and then treated with or without 0.05–10 μM of U0126, a MEK inhibitor (A–C), or 20–150 μM of DPQ, a PARP-1 inhibitor (D–F), 1 h before stimulation with 100 nM of PMA for 10 h. A and B1 represent region A and B1 of the H1R promoter, respectively. , WT promoter; , MT1 promoter. Data are presented as mean ± S.E.M. [n = 4 (A, B, and D), 7 (C and F), or 3 (E)]. **, p < 0.05 vs. control.