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. Author manuscript; available in PMC: 2013 Jul 1.
Published in final edited form as: J Med Virol. 2012 Jul;84(7):1106–1114. doi: 10.1002/jmv.23302

TABLE I.

Population Distribution of ITPA Deficiency

rs1127354 rs7270101 Predicted
ITPase activity
Predicted
ITPase deficiency
Overall,
n (%)
HCV,
n (%)
HIV/HCV,
n (%)
Wild type (C/C) Wild type (A/A) 100 83 (70.3) 35 (63.6) 48 (76.2)
Wild type (C/C) Heterozygous (A/C) 60 + 14 (11.9) 8 (14.5) 6 (9.5)
Heterozygous (C/A) Wild type (A/A) 30 ++ 14 (11.9) 6 (10.9) 8 (12.7)
Wild type (C/C) Homozygous (C/C) 30 ++ 5 (4.2) 4 (7.3) 1 (1.6)
Heterozygous (C/A) Heterozygous (A/C) 10 +++ 2 (1.7) 2 (3.6) 0
Homozygous (A/A) Wild type (A/A) <5 +++ 0 0 0

Severity of ITPase deficiency was predicted as absent, representing wild-type activity (−) and mild (+), moderate (++), or severe (+++) deficiency according to previous studies. ITPase activity was defined as the accumulated presence of minor alleles at the respective polymorphic sites [the minor alleles for both SNPs never occurred together on the same chromosome].

The prevalence of different alleles and the predicted levels of ITPAse deficiency according to the haplotypes.