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. Author manuscript; available in PMC: 2013 May 21.
Published in final edited form as: Neuron. 2012 Nov 21;76(4):721–734. doi: 10.1016/j.neuron.2012.09.018

Figure 5. Genetic Interactions among Sema-1a, pebble, and RhoGAPp190 Mutants.

Figure 5

Premature ISNb branching phenotypes (open bars) and total ISNb defects (closed bars) in embryos with indicated genotypes (*p<0.001 and †p<0.01, chi-square test). Total ISNb defects in embryos transheterozygous for Sema-1a and pbl are greater than those observed in either Sema-1a or pbl heterozygous embryos. Removing one copy of Sema-1a significantly suppresses p190 knockdown–mediated premature ISNb branching phenotypes and also total ISNb defects. Removing one copy of pbl also leads to significant suppression of p190 knockdown–mediated premature ISNb branching phenotypes. n=the number of abdominal hemisegments scored for each genotype.