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. 2011 Dec 7;138(3):377–385. doi: 10.1007/s00432-011-1103-0

Table 2.

The associations between genotypes of genes in PI3K and RAS/RAF pathways and risk of endometrial cancer

Genotypes Cases Controls Crude OR (95% CI) Adjusted OR (95% CI)* P*
AKT1 rs2498801 n = 114 n = 229
AA 48 99 1.00 1.00
AG 45 106 0.88 (0.54–1.43) 0.90 (0.55–1.48) 0.669
GG 21 24 1.81 (0.92–3.56) 1.83 (0.92–3.65) 0.086
AA/AG 93 205 1.00 1.00
GG 21 24 1.93 (1.02–3.64) 1.94 (1.02–3.67) 0.043
PIK3CA rs6443624 n = 114 n = 227
CC 73 120 1.00
CA 34 87 0.64 (0.39–1.05) 0.63 (0.39–1.04) 0.071
AA 7 20 0.58 (0.23–1.43) 0.58 (0.23–1.47) 0.252
CC 73 120 1.00 1.00
CA/AA 41 107 0.63 (0.40–1.00) 0.62 (0.39–1.00) 0.049
PIK3CA rs9838411 n = 115 n = 229
GG 81 134 1.00 1.00
GA 29 82 0.59 (0.35–0.97) 0.58 (0.35–0.96) 0.035
AA 5 13 0.64 (0.22–1.85) 0.65 (0.21–1.97) 0.443
GG 81 134 1.00 1.00
GA/AA 34 95 0.59 (0.37–0.96) 0.59 (0.36–0.95) 0.032
Combined effect (favorable alleles) n = 113 n = 227
0–1 46 65 1.00 1.00
2 35 67 0.74 (0.43–1.29) 0.77 (0.43–1.35) 0.354
>2 32 95 0.48 (0.28–0.82) 0.48 (0.27–0.83) 0.009
P trend 0.008 0.009

* Logistic regression models adjusted for age, ethnicity, and smoking status

The combined effects were grouped according to numbers of favorable alleles (rs2498801 A, rs6443624 A, and rs9838411 A were considered as favorable alleles)