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. Author manuscript; available in PMC: 2014 Jan 1.
Published in final edited form as: Shock. 2013 Jan;39(1):3–10. doi: 10.1097/SHK.0b013e3182764fe8

Fig. 3. Effect of Wnt agonist on tissue damage and cellular architecture in the liver after hepatic I/R.

Fig. 3

Histological findings of the liver in the sham (A), vehicle (B), and Wnt agonist-treated (C, WntAg) groups. Liver tissues were harvested 24 h after reperfusion, processed, and stained with hematoxylin-eosin. Representative photomicrographs at 100× magnification with 200x magnification insets. Bar length equal to 100 micrometers. (D) Semi-quantitative histologic injury score measuring differences in cytoplasmic vacuolization, cytoplasmic faiding, nuclear condensation, nuclear faiding, nuclear fragmentation, and erythrocyte stasis examined on standard hematoxylin-eosin staining as described in Materials and Methods. Data presented as means ± SE (n=4-6/group) and compared by one-way ANOVA and SNK method;*P > 0.05 vs. Sham; #P > 0.05 vs. Vehicle.