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. Author manuscript; available in PMC: 2014 Jun 1.
Published in final edited form as: J Nutr Biochem. 2012 Sep 17;24(6):1019–1027. doi: 10.1016/j.jnutbio.2012.07.010

Figure 6.

Figure 6

Effect of ABA treatment on pulmonary gene expression following infection with influenza virus.(A) PPAR γ, (B) LANCL2, (C) angiopoietin-like 4, (D) iPLA2, (E) LOX5, (F) FLAP, (G) IL-10 and (H) TNF-α. WT or myeloid KO mice were challenged with 5 × 104 TCID50 of influenza A/Udorn (H3N2) and then treated with either PBS or ABA (100 mg/ABA kg body weight) by oral gavage for 10 days. Asterisks denote statistically significant differences between the ABA-treated WT and the myeloid KO mice. Pound signs denote statistically significant differences between the ABA-treated WT and the control WT (n=10 mice per treatment and genotype).