Table 1.
Each of the four models is based on the same representation of the circulatory subsystem (1) and combines two regulatory mechanisms: one for arterial resistances (AR1 or AR2) and one for sodium excretion (SE1 or SE2).
Each of the four models is based on the same representation of the circulatory subsystem (1) and combines two regulatory mechanisms: one for arterial resistances (AR1 or AR2) and one for sodium excretion (SE1 or SE2).