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. Author manuscript; available in PMC: 2013 May 21.
Published in final edited form as: Cell. 2012 Nov 21;151(5):1042–1054. doi: 10.1016/j.cell.2012.10.044

Figure 5. Tae2 is responsible for a distinct Hsf1 branch that monitors translation stress.

Figure 5

(A) Hsf1 activity in deletions strains for the RQC and a ribosomal subunit causing synergistic activation of Hsf1. (B) GFP levels of the cotranslationally degraded reporter construct in selected strain backgrounds from (A). (C) Effect of TAE2 deletion on Hsf1 activity arising from non-translation stresses. Also see Figure S6.