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. 2012 Jul;158(Pt 7):1867–1873. doi: 10.1099/mic.0.058222-0

Fig. 1.

Fig. 1.

Susceptibility of EPS-I to complement-mediated killing. Cells (104 c.f.u.) of the wild-type strains CTG38 and CTG-R5, the CTG1291 and CTG1701 mutants that lack EPS-I, and the complemented mutant CTG1701-C were assayed for complement susceptibility. Data represent multiple experiments and the total number of samples assayed for each strain was 27 for CTG1701-C and 36 for all other strains. Means with the same number of asterisks were not significantly different from each other, but there were significant differences between means with a different number of asterisks. Error bars, sem.