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. 2012 Oct 26;304(2):H195–H205. doi: 10.1152/ajpheart.00188.2012

Fig. 1.

Fig. 1.

Chronic hypoxia (CH) had little effect on the total pulmonary vascular compliance (CL) but resulted in lower arterial compliance (Ca) and venous compliance (Cv). Nitric oxide (NO) resulted in a significant increase in compliance in CH lungs after KCl-induced vasoconstriction. Vascular compliance was estimated from vascular occlusion maneuvers in isolated perfused lungs using a five-compartment model that included CL (A), Ca (B), Cv (C), and microvascular complicance (Cc; D). Vascular occlusions were performed under four conditions, each lasting 30 min: baseline (BL), 3 mM Nω-nitro-l-arginine methyl ester (l-NAME), 30 mM KCl, and 100 μM diethylenetriamine (DETA)-NONOate. *Different from BL in the same group (P < 0.05); $different from 30 mM KCl in the same group (P < 0.05); #CH different from normoxia (N) with the same perfusate (P < 0.05).