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. Author manuscript; available in PMC: 2013 Jan 16.

Published in final edited form as: J Neurochem. 2010 Aug 25;115(2):325–332. doi: 10.1111/j.1471-4159.2010.06922.x

Fig. 4 — A single METH administration decreases both the velocity and magnitude of K⁺-stimulated DA release from striatal suspensions (A) and DA content in the VMAT-2M- and VMAT-2C-containing vesicle fractions (B). Rats received an injection of METH (15 mg/kg, s.c.) or saline vehicle (1 ml/kg, s.c.) and were killed 1 h later. Each column represents the mean ± S.E.M. of 6 to 8 independent determinations. *, indicates a statistical difference between saline- and METH-treated animals.

Fig. 4 — A single METH administration decreases both the velocity and magnitude of K⁺-stimulated DA release from striatal suspensions (A) and DA content in the VMAT-2M- and VMAT-2C-containing vesicle fractions (B). Rats received an injection of METH (15 mg/kg, s.c.) or saline vehicle (1 ml/kg, s.c.) and were killed 1 h later. Each column represents the mean ± S.E.M. of 6 to 8 independent determinations. *, indicates a statistical difference between saline- and METH-treated animals.