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Effects of hypoxia on VEGF production and cell viability of nonviral engineered ADSCs. (a) Hypoxia (1% O2 for 48 h) enhanced VEGF release from nonviral engineered ADSCs in a dose-dependent manner. (b) Hypoxia treatment led to an increase in ADSC viability in most PBAE-transfected groups, except in the groups transfected with 6 μg PBAE/DNA or Lipo.
