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Prolactin-induced ERK5 activation is independent of STAT5. A, time course of prolactin-induced STAT5 phosphorylation and activation in SVZ-aNPCs. B, no crosstalk between ERK5 and STAT5 signaling pathways. SVZ-aNPCs were pretreated for 1 h with or without a STAT5 inhibitor (STAT5-I) or MEK5 inhibitor (MEK5-I, BIX02188) prior to prolactin stimulation. β-Actin was used as a loading control. NS, no stimulation.
