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. Author manuscript; available in PMC: 2013 Oct 1.
Published in final edited form as: Muscle Nerve. 2012 Oct;46(4):482–489. doi: 10.1002/mus.23402

Figure 2.

Figure 2

90% to 5% relaxation times over sequential handgrips 1–6 for different mutation types. A. Subjects with dominant chloride channel mutations (n=11) show an increased 1st handgrip relaxation times with a pattern of warm-up of myotonia in subsequent handgrips. B. Recessive chloride channel subjects (n=10) show increased relaxations times compared to dominant, there is a pattern of warm-up in successive handgrips. C. Sodium subjects with the T1313M mutation (n=11) show paradoxical myotonia in successive handgrips. D. Subjects with R1448H mutations (n=5), on the other hand have essentially normal QMA relaxation times. E. No clear myotonia was demonstrated in subjects with G1306A mutations (n=5). F. Although there were no clear abnormalities on QMA for DM2 (n=6), in the group there is a subtle trend towards warm-up.