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Proposed models for SUMO1 regulation of lamin A tails. Proposed mechanisms by which SUMO1 modification of lamin A tails might regulate 1) tissue-appropriate binding or release of SREBP1 (or other partners) and 2) recruitment of hypothetical SIM-containing partner(s) relevant to adipose tissue. We propose that both types of interaction are misregulated in a subset of FPLD patients.
