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. 2013 Jan 10;10:3. doi: 10.1186/1742-4690-10-3

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Copyright ©2013 Fouda et al.; licensee BioMed Central Ltd.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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No difference in binding to colonic epithelial cells or galactosyl ceramide (Galcer) between Env variants from women who did and did not postnatally transmit HIV-1 to their infants. Binding of milk Env variants from postnatal-transmitting (A) and nontransmitting (B) mothers to colonic epithelial HT-29 cells. (C) Binding of gp120 proteins expressed by milk Env virus variants to Galcer measured by biolayer interferometry. (D) Binding of gp120 variants from transmitting mothers to Galcer. Stars represent transmitted variants, error bars represent standard deviation and lines represent means of two experiments performed in triplicate.