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. Author manuscript; available in PMC: 2013 Oct 1.
Published in final edited form as: Mol Immunol. 2012 Jun 27;52(3-4):207–216. doi: 10.1016/j.molimm.2012.05.018

Fig. 3.

Fig. 3

Human β2-GPI attenuates IR-induced intestinal damage and PGE2 production in a dose dependent manner. Mouse β2-GPI (200 μg/kg) or Human β2-GPI (H) at 200, 40 or 20 μg/kg was administered prior to subjecting C57Bl/6 mice to Sham or IR treatment. Two hours after initiation of reperfusion, mid-jejunal intestinal sections (75–150 villi per animal) were scored (A) and ex vivo PGE2 (B) production were assessed. Each bar represents 4–10 animals. * = p ≤ 0.05 compared to Sham, φ = p ≤ 0.05 compared to I/R treatment animals not receiving β2-GPI.