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. 2012 Dec 18;168(1):238–252. doi: 10.1111/j.1476-5381.2012.02176.x

Table 4.

The extent of LV dysfunction following I–R injury was not affected by sex, in either hearts isolated from annexin-A1 wild-type or ANX-A1−/− mice ex vivo

LVDP (% baseline, min post reperfusion)
Experimental group 0 1 2 5 10 20 30 40 AUC x 103 n
Wild-type male I–R 51 ± 19 94 ± 27 57 ± 9 53 ± 9 50 ± 9 56 ± 11 63 ± 12 65 ± 13 2.33 ± 0.43 7
Wild-type female I–R 34 ± 9 75 ± 13 60 ± 7 45 ± 9 44 ± 13 60 ± 9 65 ± 8 63 ± 5 2.27 ± 0.31 4
ANX-A1−/− male I–R 27 ± 4 56 ± 85 37 ± 6 10 ± 35 13 ± 35 28 ± 6 29 ± 7 29 ± 75 1.00 ± 0.205 8
ANX-A1−/− female I–R 17 ± 5 57 ± 9 33 ± 9 16 ± 5 21 ± 7 30 ± 6 33 ± 5 34 ± 5 1.13 ± 0.20 6

Analysis of the time course of recovery of LVDP over 40 min of reperfusion on two-way anova indicates that P(min post reperfusion) < 0.0001; P(experimental group) < 0.0001; P(interaction) = NS. Analysis of AUC for LVDP on two-way anova indicates that P(gender) = NS; P(experimental group) < 0.001; P(interaction) = NS.

On two-way anova with SNK post hoc analysis, hearts from male ANX-A1−/− have poorer recovery from I–R injury than male wild-type mice (P < 0.01). No significant differences were observed between male and female mice of either genotype.