Skip to main content
. Author manuscript; available in PMC: 2014 Feb 7.
Published in final edited form as: Mol Cell. 2012 Dec 20;49(3):464–473. doi: 10.1016/j.molcel.2012.11.017

Figure 4. Hsp90Ec mutants are defective in client binding.

Figure 4

(A) The ATPase activity of Hsp90Ec wild type or mutants with or without L2 was measured. Data from 3 replicates are presented as mean ± SEM.

(B) Binding of Hsp90Ec wild type or mutants to IAEDANS-labeled Δ131Δ was measured by fluorescence anisotropy. Binding curves are the average of two independent measurements.

See also Figure S4.

HHS Vulnerability Disclosure