. 2013 Feb 5;138(3):208–215. doi: 10.1111/imm.12029

Table 3.

Incidence and severity experimental autoimmune encephalomyelitis (EAE) induced following clinically silent inflammation of the cerebellum

Transferred cells¹	Incidence of nEAE² (peak CS ± SD)	Incidence of cEAE² (peak CS ± SD)
Day 0: 1 × 10⁶ IFN‐γ^−/−	1/20 (2)	0/20
Day 0: 1 × 10⁶ IFN‐γ^−/− Day 5: WT	16/20 (3·4 ± 1·1)	20/20 (2·8 ± 1·0)
Day 0: 1 × 10⁶ IFN‐γ^−/− Day 5: OVA	5/20 (1·8 ± 0·4)	0/20
Day 0: 1 × 10⁶ IFN‐γ^−/− Day 23: WT	6/20 (2·6 ± 0·8)	20/20 (3·0 ± 1·0)
Day 0: 1 × 10⁶ IFN‐γ^−/− Day 23: OVA	1/20 (1)	0/20

Transferred cells: 1 × 10⁶ interferon‐γ (IFN‐γ) ‐deficient MOG_35–55‐specific CD4⁺ T cells (IFN‐γ^−/−) were administered at the start of the experiment then either 5 or 23 days later mice received 5 × 10⁶ wild‐type MOG_35–55‐specific CD4⁺ T cells (WT) or 5 × 10⁶ wild‐type ovalbumin‐specific CD4⁺ T cells (OVA) (where MOG_35–55 is a peptide comprising amino acids 35 to 55 of the myelin oligodendrocyte glycoprotein).

(Peak CS ± SD): The mean peak clincial score and standard deviations for each group are shown.