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. 2013 Feb 1;10:6. doi: 10.1186/1742-4682-10-6

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Copyright ©2013 Aller et al.; licensee BioMed Central Ltd.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Surgical stressful inflammatory response. The host organism provides the inflamed interstitium molecules and cells by up-regulating a neurogenic-related axis (NA), a bone marrow-related axis (BMA) and a gut-liver axis (GLA). The molecular and cellular infiltration of the “endothelial egg” induces the development of a new tissue or organ using inflammatory mechanisms. BCE: blood capillary endothelium; C: coagulation; E: epithelium; ESC: endothelial precursor cell; F: fibroblast; HEVE: high endothelial venule endothelium; HSC: hemopoietic stem cell; LE: lymphatic endothelium; Leu: leukocytes; M: microbiome; MSC: mesenchymal stem cell; PCVE: postcapillary venule endothelium; SC: resident stem cell; SG: suprarenal gland.