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. Author manuscript; available in PMC: 2013 Feb 25.
Published in final edited form as: J Med Primatol. 2008 Dec;37(Suppl 2):46–54. doi: 10.1111/j.1600-0684.2008.00327.x

Fig. 2.

Fig. 2

CXCL12 mRNA expression in ALN tissues of cynomolgus macaques during SIV infection. (A–C) ISH was performed for CXCL12 mRNA in axillary LN tissue sections from uninfected (A), acutely SIV-infected (B) and AIDS-developing macaques (C). (D–F) ISH and immunohistochemistry staining were performed simultaneously to detect the cellular sources of CXCL12 in axillary LN tissue sections; (D) CXCL12 ISH and DC-SIGN IHC; (E) CXCL12 ISH and CD68 IHC; (F) CXCL12 ISH and CD3 IHC. Parallel hybridization of tissue sections with the cognate sense control probe provided no specific ISH signal and parallel staining of tissue sections with the cognate isotype control provided no specific IHC signal (not shown). Original magnifications, ×200 (A–C) and ×600 (D–F).