Neuroprotective effects of SUN11602 and bFGF against glutamate-induced
toxicity in primary cultures of cerebrocortical neurons from WT and
Calb–/– mice. These data indicate that calbindin-D28k
in WT mice may play important roles in the neuroprotective mechanisms
by SUN11602 and bFGF. In Calb–/– mice, the
molecular function of calbindin-D28k was mostly diminished, although
there was a slight improvement in neuroprotection with higher concentrations
of SUN11602. Following the 24 h incubation with SUN11602 and bFGF,
the cultures were exposed to either 100 or 50 μM glutamate for
another 24 h, and cell viability was then determined by MTT assay.
The toxic concentrations of glutamate (100 or 50 μM) were employed
for which those concentrations were suitable for the cultures in order
to react sensitively and appropriately. *p < 0.05,
**p < 0.01, ***p < 0.001 compared
to the glutamate group (means ± SEM, n = 6,
Dunnett’s multiple comparison test).