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. 2012 Sep 24;22(6):845–854. doi: 10.1089/scd.2012.0395

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Copyright 2013, Mary Ann Liebert, Inc.

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FIG. 2. — hucMSC-Ex promoted the recovery of carbon tetrachloride (CCl₄)-induced mouse liver injury. (A) hucMSC-Ex location in mouse liver by an in vivo imaging system. (a) The phosphate-buffered saline (PBS) group liver; (b) The cell dye-labeled hucMSC-Ex administrated into mouse liver showed red fluorescence. (B) Pictures of mouse livers. (a) normal group; (b) CCl₄ group, 6 weeks after CCl₄ injury; (c) PBS group 3 weeks after PBS treatment; (d) hucMSCs-Ex group 3 weeks after hucMSC-Ex transplantation. The arrows indicated the liver damage/fibrotic areas. (C) Hematoxylin and eosin dyeing of liver sections (200×). (a) normal group; (b) PBS group, 1 week; (c) PBS group, 2 weeks; (d) PBS group, 3 weeks; (e) hucMSC-Ex group, 1 week; (f) hucMSC-Ex group, 2 weeks; (g) hucMSC-Ex group, 3 weeks. (D) Serum hyaluronic acid (HA) level 3 weeks after hucMSC-Ex transplantation. (E) Serum transforming growth factor (TGF)-β1 level 3 weeks after hucMSC-Ex transplantation. (F) Serum alanine aminotransferase (ALT) and aspartate aminotransferase (AST) levels after hucMSC-Ex transplantation. *P<0.05, **P<0.01, and ***P<0.001.