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. 2013 Mar 7;9(3):e1003324. doi: 10.1371/journal.pgen.1003324

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© 2013 Xie et al

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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A. Survival rates of WT and loss- and gain-of-function mutants of tir-1 fed PA14 under standard slow killing pathogenesis assay conditions. B. daf-19 mutants displayed elevated susceptibility to killing by PA14. C. Survival rates of vector control and daf-19 RNAi-treated worms fed PA14. D. Transgenic expression of daf-19 in the intestine by the ges-1 promoter partially rescued the elevated PA14 susceptibility of daf-19 mutants. E. daf-19 deficiency suppressed the enhanced PA14 resistance of tir-1(yz68gf) mutants. F. daf-19;atf-7(lf) mutants displayed elevated susceptibility to killing by PA14 similarly to atf-7(lf) mutants. The fraction of live animals was determined at each indicated time points. The experiments were performed in triplicates. Results are representative of at least two independent experiments.