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. 2013 Mar 13;8(3):e58574. doi: 10.1371/journal.pone.0058574

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© 2013 Huang et al

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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Sections of kidney from DN patients were stained with anti-GLIPR-2 (GLIPR-2), anti-p-ERK1/2 (P-ERK), anti-E-cadherin (E-ca), anti-vimentin (VIMENTIN) and anti-α-smooth muscle actin (α-SMA) antibodies. The GLIPR-2 staining was increased in PTCs of DN kidney (A) compared with normal kidney (B). In addition, p-ERK1/2 was also increased in the PTCs of DN kidney (C and D). The EMT marker E-cadherin decreased but the EMT markers vimentin and α-SMA increased in the DN kidney compared with control (E to J).