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Exploitation of host cell lipid phosphatidylinositol (PI) derivatives to anchor Legionella pneumophila effectors. Many defects in organelle trafficking/intracellular multiplication (Dot/Icm)-translocated effectors such as SidC, SidM, LidA and LpnE are anchored to the Legionella-containing vacuole (LCV) membrane through PI(4)P or PI(3)P to interfere with signal transduction and promote host vesicle trafficking.
