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. Author manuscript; available in PMC: 2013 Apr 1.
Published in final edited form as: Kidney Int. 2011 Jan 12;79(8):883–896. doi: 10.1038/ki.2010.526

Figure 3. Insulin-induced p-eNOS and p-GSKα in the glomeruli and tubules of diabetic and control SD rats and ZL and ZF rats.

Figure 3

A and B. Representative immunoblots of p-eNOS from glomerular proteins. Data from three experiments were quantitated by densitometry. A) Cont. vs. DM, B) ZL vs. ZF, n=6 in each group, *P<0.05, **P<0.001.

C and D. Representative immunoblots of p-GSKα from glomerular and tubular fractions. Data from three experiments were quantitated by densitometry. C) Cont. vs. DM, D) ZL vs. ZF, n=6 in each group, **P<0.001.

E and F. Representative immunoblots of p-GSKβ from glomerular and tubular fractions. Data from three experiments were quantitated by densitometry. E) Cont. vs. DM, F) ZL vs. ZF, n=6 in each group, **P<0.001. These data are expressed as means ± SD. Cont.= control rats, DM=STZ-induced diabetic SD rats, ZL=Zucker lean rats, ZF=Zucker fatty rats, G=glomeruli, T=tubules.

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