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. 2013 Mar 18;110(14):5522–5527. doi: 10.1073/pnas.1214467110

Fig. 2.

Fig. 2.

Ascarosides increase thermotolerance at 35 °C in wild-type (N2) C. elegans in a concentration-dependent manner. (A) Survivorship of adult worms at 35 °C exposed to 2 nM, 200 nM, and 1 µM ascr#2; 2 nM ascr#2: n = 278, m = 10.6 h, P < 0.0001; 200 nM ascr#2: n = 313, m = 11.6 h, P < 0.0001; 1 µM ascr#2: n = 142, m = 12.1 h, P < 0.0001; and mock-treated control: n = 1062, m = 9.7 h. (B) Survivorship of adult worms at 35 °C exposed to 2 nM, 200 nM, and 1 µM ascr#2; 2 nM ascr#3: n = 278, m = 9.8 h, P = 0.004; 200 nM ascr#3: n = 289, m = 12.6 h, P < 0.0001; 1 µM ascr#3: n = 126, m = 12.6 h, P < 0.0001; and mock-treated control: n = 1062, m = 9.7 h. (C) Survivorship of adult worms at 35 °C exposed to 400 nM ascr#2 compared with mock-treated controls, using two different nutritional conditions [adlib (with bacteria) and DD (without bacteria)]; adlib ascr#2: n = 65, m = 12.2 h, P < 0.0001; adlib control: n = 75, m = 10.2 h; DD ascr#2: n = 103, m = 12.9 h, P = 0.145; DD control: n = 103, m = 13.3 h.