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. 2013 Mar 31;22(1):31–37. doi: 10.5607/en.2013.22.1.31

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Copyright © Experimental Neurobiology 2013.

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 3 — AAD-2004 reverted the neuritic atrophy in the brain of Tg-βCTF99/B6 mice. (A) Photomicrograph showing anti-MAP-2 antibody-stained parietal cortex of non-transgenic control mouse. The rectangle is the region shown with high magnification in B-D. All animals analyzed were 18-18.5 months-old. (B-D) Photomicrographs showing anti-MAP-2 antibody-stained parietal cortex of non-transgenic control mice (WT; B), Tg-βCTF99/B6 control mice (TG; C), and Tg-βCTF99/B6 mice fed with AAD-2004 (TG+AAD-2004; D). Note the reduced numbers and morphology of long-running apical dendrites in Tg-βCTF99/B6 mice (C), while the protection was evident in the Tg-βCTF99/B6 mice fed with AAD-2004 (D). 5-8 animals were analyzed for each group. Scale bar, 200 µm.