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. Author manuscript; available in PMC: 2014 May 1.
Published in final edited form as: Immunol Rev. 2013 May;253(1):40–52. doi: 10.1111/imr.12045

Fig. 4. miR-182 is dispensable for NK cell responses to MCMV infection.

Fig. 4

(A) Equal numbers of WT (CD45.1+) and miR-182−/− (CD45.2+) Ly49H+ NK cells were co-transferred into Ly49H-deficient hosts. Following infection with MCMV, the percentage of adoptively transferred WT (black) and miR-182−/−(red) Ly49H+ NK cells within the total NK cell population in blood was assessed at the indicated time points post-infection. Error bars show SEM (n = 3). Data are representative of three independent experiments. (B) Schematic representation of the miR-183 ~ 182 cluster. miR-183, miR-96, and miR-182 share sequence homology, particularly within the seed sequence used to bind target mRNAs (pink box).