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. 2013 Apr 15;140(8):1645–1654. doi: 10.1242/dev.088013

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© 2013. Published by The Company of Biologists Ltd

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial Share Alike License (http://creativecommons.org/licenses/by-nc-sa/3.0), which permits unrestricted non-commercial use, distribution and reproduction in any medium provided that the original work is properly cited and all further distributions of the work or adaptation are subject to the same Creative Commons License terms.

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Fig. 4. — Depletion of MAP kinase pathway components suppress puf-8(-); gap-3(-) phenotype. Dissected gonads of puf-8(-); gap-3(-) young adults following the indicated RNAi treatments stained as described in Fig. 3. Although let-60(RNAi) reduces dpMPK-1 levels considerably throughout the germline, sur-6(RNAi) and rom-1(RNAi) does so in the early pachytene region. In all three cases, tumourous proliferation is abolished and gametes are seen in the proximal region - oocytes in let-60(RNAi) and sperm in sur-6(RNAi) and rom-1(RNAi). See text and Table 1 for additional information.