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. Author manuscript; available in PMC: 2013 Apr 10.
Published in final edited form as: Mol Cell Neurosci. 2007 Feb 8;35(1):57–63. doi: 10.1016/j.mcn.2007.02.003

Fig. 1.

Fig. 1

Scheme of viruses and tracing method used. (A) Schematic of the FE65 dominant-negative viral constructs used. FE65-ΔPID2 lacks the entire PID2 domain. FE65-mPID2 and FE65-mWW contain the indicated point mutations, which disrupt the interaction of FE65 with APP and Mena, respectively. (B) Example of a Neurolucida trace depicting the terms used (i.e. neurite order, branch points, etc.).