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. 2013 Feb 15;5(3):504–513. doi: 10.1093/gbe/evt026

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© The Author(s) 2013. Published by Oxford University Press on behalf of the Society for Molecular Biology and Evolution.

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0/), which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 4.— — Changes in disorder across branches. The result of the parsimony analysis of disorder to order transitions or vice versa over the phylogentic tree as changes per branch. Branches with more than 5% of sites in DOT are shown with wider lines: (A) capsid, (B) membrane glycoprotein precursor, (C) envelope, (D) NS1, (E) NS2B, (F) NS3, (G) NS5, and (H) NS4B. (For complete output, including NS2A, 2K, and NS4A, which have no branches with more than 5% of sites in DOT, see Supplementary fig. S2, Supplementary Material online.)

Fig. 4.— — Changes in disorder across branches. The result of the parsimony analysis of disorder to order transitions or vice versa over the phylogentic tree as changes per branch. Branches with more than 5% of sites in DOT are shown with wider lines: (A) capsid, (B) membrane glycoprotein precursor, (C) envelope, (D) NS1, (E) NS2B, (F) NS3, (G) NS5, and (H) NS4B. (For complete output, including NS2A, 2K, and NS4A, which have no branches with more than 5% of sites in DOT, see Supplementary fig. S2, Supplementary Material online.)