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. Author manuscript; available in PMC: 2013 Apr 12.
Published in final edited form as: Horm Behav. 2008 Aug 8;54(5):726–734. doi: 10.1016/j.yhbeh.2008.07.013

Fig. 1.

Fig. 1

Represents mean central entries in the open field for: wildtype (WT) mice (top) administered acute vehicle control (white bar; n=15), 3β-diol (horizontally-striped bar; n=11), 3α-diol (black bar; n=13), or androsterone (androst; gray-stripe bar; n=12); to ERβ receptor knockout (βERKO) mice (middle) administered acute vehicle control (white bar; n=21), 3β-diol (horizontally-striped bar; n=34), 3α-diol (black bar; n=34), or androsterone (androst; gray-stripe bar; n=29); and to rats (bottom) administered acute vehicle control (white bar; n=22), 3β-diol (horizontally-striped bar; n=26), 3α-diol (black bar; n=25), or androsterone (androst; gray-stripe bar; n=25). Rats and WT mice administered 3α-diol or 3β-diol had significantly more central entries than animals administered androsterone or vehicle. * Denotes significant difference (p<.05).