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The effect of AGS3 expression on functional desensitization of D2LDR-modulated AC1 activity. HEK293 cells stably expressing AC1, the D2LDR, and AGS3-Venus were treated with quinpirole (1 μM) or vehicle for 2 hrs, followed by selective AC1 stimulation with A23187 (10 μM) and activation of the D2LDR with quinpirole (1 μM). Data represent the mean ± S.E.M. of three independent experiments
