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. 2013 Apr 29;3:99. doi: 10.3389/fonc.2013.00099

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Copyright © 2013 Pfeiffer, Burow, Schleicher, Handgretinger and Lang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits use, distribution and reproduction in other forums, provided the original authors and source are credited and subject to any copyright notices concerning any third-party graphics etc.

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Influence of HDACi and DNMTi on NK susceptibility of MHH-CALL-4 cells. Leukemic cells were incubated with the indicated concentrations of HDACi and DNMTi for 48 h. Resting NK cells (A) or IL-2 stimulated NK cells (B) were used as effector cells. A lysis-ratio was calculated from each experiment as following: specific lysis with HDACi/DNMTi/specific lysis without HDACi/DNMTi. Shown are mean values and standard deviation from an effector-to-target cell ratio of 20:1 [n = 6 for vorinostat (four different donors), n = 15 for valproic acid (six different donors), n = 7 for decitabine (four different donors), n = 4 for azacytidine (four different donors), **p < 0.01].