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. Author manuscript; available in PMC: 2013 May 2.
Published in final edited form as: Psychoneuroendocrinology. 2008 Apr 2;33(5):659–669. doi: 10.1016/j.psyneuen.2008.02.006

Figure 4.

Figure 4

Exposure to CVS elevated basal AM plasma corticosterone levels (A). Lesions of the BSTpm potentiated plasma corticosterone response to restraint at 20 min and 40 min following onset of stress, regardless of CVS exposure (B). Exposure to CVS elevated the plasma corticosterone response at 40 min following onset of restraint only in rats with lesions. Lesions of the BSTpm enhanced the integrated plasma corticosterone response regardless of CVS exposure (C). Additionally, exposure to CVS elevated the integrated plasma corticosterone response only in rats with lesions (C). Data are presented as mean ± SEM, n = 8–12. * p < 0.05 versus respective ‘No CVS’, # p < 0.05 versus respective ‘Sham’. Abbreviations: AUC, area under the curve.